Control Central Car Panel Accessor Car 1998-2005 E46 Series 3 BMW for Trim Cover Car & Truck Interior Trim


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Car Central Control Panel Cover Trim for BMW 3 Series E46 1998-2005 Car Accessor
Condition: New MPN:

Does not apply

Other Part Number: Does Not Apply Brand:

SODIAL

Interchange Part Number: Universal Marke: SODIAL
Non-Domestic Product: No Hersteller: SODIAL
Fitment Type: Custom Type: Regular Type
Number of Pieces: Universal Modification Description: No
Custom Bundle: No Country/Region of Manufacture: China
Modified Item: No Surface Finish: Universal
Superseded Part Number: Universal Applicable Regions: Universal
Warranty: 1 Year Color:

Black

Bundle Description: No Manufacturer Part Number:

Does Not Apply

Mounting Hardware Included: No Material: Universal
Placement on Vehicle: Universal UPC:

194452877591

ISBN:

194452877591

EAN:

194452877591

published on tue nov 09 2021

Control Central Car Panel Accessor Car 1998-2005 E46 Series 3 BMW for Trim Cover Car & Truck Interior Trim

gandhi, s., klein, j., robertson, a., pena-hernandez, m. a.,

Control Central Car Panel Accessor Car 1998-2005 E46 Series 3 BMW for Trim Cover Car & Truck Interior Trim

lin, m. j., roychoudhury, p., lu, p., fournier, j., ferguson, d., mohamed bakhash, s. a., muenker, m. c., srivathsan, a., wunder, e. a., kerantzas, n., wang, w., pyle, a., wilen, c. b., ogbuagu, o., greninger ,, a. l., iwasaki, a., schulz, w. l., ko, a. i.

sars-cov-2 remdesivir resistance mutations have been generated in vitro but have not been reported in patients receiving treatment with the antiviral agent. we present a case of an immunocompromised patient with acquired b-cell deficiency who developed an indolent, protracted course of sars-cov-2 infection. remdesivir therapy alleviated symptoms and produced a transient virologic response, but her course was complicated by recrudescence of high-grade viral shedding. whole genome sequencing identified a mutation, e802d, in the nsp12 rna-dependent rna polymerase which was not present in pre-treatment specimens. in vitro experiments demonstrated that the mutation conferred a ~6-fold increase in remdesivir ic50 but resulted in a fitness cost in the absence of remdesivir. sustained clinical and virologic response was achieved after treatment with casirivimab-imdevimab. although the fitness cost observed in vitro may limit the risk posed by e802d,

, this case illustrates the importance of monitoring for remdesivir resistance and the potential benefit of combinatorial therapies in immunocompromised patients with sars-cov-2 infection.

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